Vili
Mostrando 1-8 de 8 artigos, teses e dissertações.
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1. Hypercapnia attenuates ventilator-induced lung injury through vagus nerve activation
Abstract Purpose: To investigate the role of vagus nerve activation in the protective effects of hypercapnia in ventilator-induced lung injury (VILI) rats. Methods: Male Sprague-Dawley rats were randomized to either high-tidal volume or low-tidal volume ventilation (control) and monitored for 4h. The high-tidal volume group was further divided into either
Acta Cir. Bras.. Publicado em: 25/11/2019
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2. Fotografia cervical digital para rastreamento de câncer de colo uterino e suas lesões precursoras
Background: O câncer de colo uterino é um grande problema de saúde pública no Brasil e no mundo. Atinge cerca de meio milhão de mulheres, sendo que 50 % destas morrem. Os métodos visuais, alternativos á citologia e viáveis para países em desenvolvimento, têm sido avaliados mais intensamente na última década. Objetivos: Avaliar o desempenho da Fot
Publicado em: 2010
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3. Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos / Immunohistochemical evaluation of the cytoskeletal alterations in the alveolar wall in an experimental model of ventilator-induced lung injury in rats
INTRODUÇÃO: A ventilação mecânica é uma terapia importante, mas com possíveis complicações. Uma das mais relevantes é a lesão pulmonar induzida pelo ventilador (VILI do inglês Ventilator-induced lung injury). Devido à hiperdistensão alveolar, o pulmão inicia um processo inflamatório, com infiltrado neutrofílico, formação de membrana hialin
Publicado em: 2009
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4. Effects of recruitment maneuver during expiration and inspiration analyzed by thoracic CT scan in patients with acute lung injury and acute respiratory distress syndrome / Efeitos da manobra de recrutamento alveolar nas fases inspiratória e expiratória na tomografia computadorizada de tórax em pacientes com lesão pulmonar aguda ou síndrome do desconforto respiratório agudo
The goal of Maximal Recruitment Strategy (MRS) guided by thoracic CT scan is to minimize alveolar collapse and the mechanisms of ventilator induced lung injury (VILI). The objectives of this study were to compare by quantitative analyzes of CT scan image of the lungs obtained during MRS of patients with ARDS, the following parameters: collapse, overdistensio
Publicado em: 2008
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5. Critical role for CXCR2 and CXCR2 ligands during the pathogenesis of ventilator-induced lung injury
Mortality related to adult respiratory distress syndrome (ARDS) ranges from 35% to 65%. Lung-protective ventilator strategies can reduce mortality during ARDS. The protective strategies limit tidal volumes and peak pressures while maximizing positive end-expiratory pressure. The efficacy of this approach is due to a reduction of shear-stress of the lung and
American Society for Clinical Investigation.
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6. Protective ventilation of patients with acute respiratory distress syndrome
In a recent issue of the British Journal of Anaesthesia, Moloney and Griffiths reviewed clinically pertinent issues surrounding the management of the acute respiratory distress syndrome (ARDS) patient, particularly as it pertains to the treatment of ventilator induced/associated lung injury (VILI). In addition to highlighting the important observations that
BioMed Central.
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7. GADD45a is a novel candidate gene in inflammatory lung injury via influences on Akt signaling
We explored the mechanistic involvement of the growth arrest and DNA damage-inducible gene GADD45a in lipopolysaccharide (LPS)- and ventilator-induced inflammatory lung injury (VILI). Multiple biochemical and genomic parameters of inflammatory lung injury indicated that GADD45a−/− mice are modestly susceptible to intratracheal LPS-induced lung injury and
The Federation of American Societies for Experimental Biology.
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8. Activated protein C protects against ventilator-induced pulmonary capillary leak
The coagulation system is central to the pathophysiology of acute lung injury. We have previously demonstrated that the anticoagulant activated protein C (APC) prevents increased endothelial permeability in response to edemagenic agonists in endothelial cells and that this protection is dependent on the endothelial protein C receptor (EPCR). We currently inv
American Physiological Society.