Tanning Agents
Mostrando 1-5 de 5 artigos, teses e dissertações.
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1. Remoção da adstringência de frutos de caquizeiro 'Giombo' sob diferentes temperaturas.
O objetivo deste trabalho foi avaliar o efeito de diferentes temperaturas durante o período de exposição ao vapor de álcool etílico sobre a qualidade dos frutos de caquizeiro cultivar Giombo. Os frutos foram expostos ao vapor de álcool etílico durante 42 horas, sob temperaturas de 10, 20 ou 30°C e 95% de umidade relativa. As características química
Pesquisa Agropecuária Brasileira. Publicado em: 2011
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2. Dispersiones acuosas de poliuretano bloqueado: una alternativa como agente curtiente
Two types of crosslinking agents blocked polyurethanes were synthesized and evaluated as tanning agents of bovine skins. The results of the synthesis of aqueous dispersions of blocked polyurethane (DPU’s) are presented. They were based on prepolymers from the reaction of the aliphatic diisocyanates H12MDI 4,4’-methylenebis(cyclohexyl isocyanate) and HDI
Polímeros. Publicado em: 2008-06
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3. Molecular and Functional Analyses of Amino Acid Decarboxylases Involved in Cuticle Tanning in Tribolium castaneum*
Aspartate 1-decarboxylase (ADC) and 3,4-dihydroxyphenylalanine decarboxylase (DDC) provide β-alanine and dopamine used in insect cuticle tanning. β-Alanine is conjugated with dopamine to yield N-β-alanyldopamine (NBAD), a substrate for the phenol oxidase laccase that catalyzes the synthesis of cuticle protein cross-linking agents and pigment precursors. W
American Society for Biochemistry and Molecular Biology.
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4. The Usf-1 transcription factor is a novel target for the stress-responsive p38 kinase and mediates UV-induced Tyrosinase expression
The stress-activated signalling cascade leading to phosphorylation of the p38 family of kinases plays a crucial role during development and in the cellular response to a wide variety of stress-inducing agents. Although alterations in gene expression characteristic of the stress response require the regulation of key transcription factors by the p38 family, f
Oxford University Press.
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5. DNA damage enhances melanogenesis.
Although the ability of UV irradiation to induce pigmentation in vivo and in vitro is well documented, the intracellular signals that trigger this response are poorly understood. We have recently shown that increasing DNA repair after irradiation enhances UV-induced melanization. Moreover, addition of small DNA fragments, particularly thymine dinucleotides (