Saccadic Movements
Mostrando 13-24 de 50 artigos, teses e dissertações.
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13. Saccadic function in spasmodic torticollis.
Twelve patients with idiopathic spasmodic torticollis were compared with 19 normal controls on tests of saccadic eye movements thought to depend upon normal basal ganglia function. The patients were able to make random, predictive, remembered, and self-paced saccades equally as well as control subjects. This suggests that those parts of the basal ganglia whi
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14. Visually and memory guided saccades in a case of cerebellar saccadic dysmetria.
Saccades under four specific test conditions (visually guided, visually remembered, vestibular remembered, and cervical remembered) were studied in a 38 year old man with ocular dysmetria due to an angioma of the dorsal cerebellar vermis. The aim of the study was to investigate if the saccadic disorder was specific to certain subsets of saccades elicited by
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15. On the predictive control of foveal eye tracking and slow phases of optokinetic and vestibular nystagmus.
Smooth pursuit and saccadic components of foveal visual tracking as well as more involuntary ocular movements of optokinetic (o.k.n.) and vestibular nystagmus slow phase components were investigated in man, with particular attention given to their possible input-adaptive or predictive behaviour. Each component in question was isolated from the eye movement r
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16. Orienting to threat: faster localization of fearful facial expressions and body postures revealed by saccadic eye movements
Most studies investigating speeded orientation towards threat have used manual responses. By measuring orienting behaviour using eye movements a more direct and ecologically valid measure of attention can be made. Here, we used a forced-choice saccadic and manual localization task to investigate the speed of discrimination for fearful and neutral body and fa
The Royal Society.
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17. Ocular motility in alternating squints: an electro-oculographic study.
Ocular movements were studied electro-oculographically in 10 normal subjects and 10 patients with alternating strabismus. Fixation and pursuit movements and to a less extent the saccadic movements were observed to be abnormal in patients with alternating strabismus. The defect in pursuit movements was shown to be statistically significant. There was no corre
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18. Oculomotor disorders in Huntington's chorea.
Electro-oculographic recordings were obtained from 11 patients with Huntington's disease. Significant reduction of saccadic velocity was constantly found. In all the patients vertical saccades were much more impaired than horizontal. When present, vertical saccades showed long latency, low amplitude, low velocity, and disturbances related to blinking. Follow
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19. Vestibulo-ocular reflex abnormality in Vogt-Koyanagi-Harada syndrome.
The eye movements of 15 patients with Vogt-Koyanagi-Harada (VKH) syndrome were analysed. Transient dizziness occurred in 60% of the patients. Eight patients (53%) exhibited horizontal jerk nystagmus that was completely inhibited by visual fixation. Eight cases (53%) exhibited the elevation of vestibulo-ocular reflex (VOR) gain in darkness. Six of the patient
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20. Functional mapping of the human cerebellum with positron emission tomography.
Alterations of local neuronal activity induced within the human cerebellum by tactile stimulation and voluntary movement were mapped with positron emission tomographic measurements of brain blood flow. Finger movements produced bilateral, parasagittal blood-flow increases in anterior, superior hemispheric cortex of the cerebellum. Responses to tactile finger
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21. Binocular co-ordination of human vertical saccadic eye movements.
1. The binocular co-ordination of human vertical saccades was analysed systematically over the full oculomotor range, with a precise and accurate scleral sensor coil technique. Effects of amplitude (1.25-70 deg), direction (upward vs. downward and centripetal vs. centrifugal), as well as position (upper or lower sector of vertical oculomotor range), were inv
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22. The time course of visual information accrual guiding eye movement decisions
Saccadic eye movements are the result of neural decisions about where to move the eyes. These decisions are based on visual information accumulated before the saccade; however, during an ≈100-ms interval immediately before the initiation of an eye movement, new visual information cannot influence the decision. Does the brain simply ignore information prese
National Academy of Sciences.
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23. Long-lasting modifications of saccadic eye movements following adaptation induced in the double-step target paradigm
The adaptation of saccadic eye movements to environmental changes occurring throughout life is a good model of motor learning and motor memory. Numerous studies have analyzed the behavioral properties and neural substrate of oculomotor learning in short-term saccadic adaptation protocols, but to our knowledge, none have tested the persistence of the oculomot
Cold Spring Harbor Laboratory Press.
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24. Oculomotor control in Gilles de la Tourette syndrome.
Saccadic eye movements, fixation and smooth pursuit were studied in 28 children with Gilles de la Tourette syndrome and found to be normal. A link has been postulated between Gilles de la Tourette syndrome and other movement disorders. The results obtained in the present series do not support this hypothesis.