Programed Death Cell
Mostrando 1-4 de 4 artigos, teses e dissertações.
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1. In vitro cytotoxicity of chemical preservatives on human fibroblast cells
ABSTRACT Preservatives are widely used substances that are commonly added to various cosmetic and pharmaceutical products to prevent or inhibit microbial growth. In this study, we compared the in vitro cytotoxicity of different types of currently used preservatives, including methylparaben, imidazolidinyl urea (IMU), and sodium benzoate, using the human newb
Braz. J. Pharm. Sci.. Publicado em: 14/05/2018
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2. Efeitos agudos do exercício resistido sobre marcadores da resposta inflamatória e imune
O objetivo dos pesquisadores do presente estudo foi examinar os efeitos agudos do Exercício Resistido (ER) sobre a apoptose (Anexina V+) e a migração (CX3CR1) de linfócitos T CD4+ e CD8+. 12 sujeitos adultos realizaram duas sessões de ER (3 séries em 9 exercícios) com 1 minuto (Hiper-1) e 3 minutos (Hiper-3) de intervalo entre as séries e exercícios
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 11/12/2012
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3. Estresse osmótico e do retículo endoplasmático induzem morte celular programada de maneira dependente das proteínas NRPs / Osmotic- and ER-stress induce NRPs-dependent programed cell death
The identification of cell signaling pathways in response to different stresses and the interactions among these pathways have become major focus for understanding the molecular bases of plant cell-environment interactions. Recently, a novel integrative pathway between ER- and osmotic-stress has been described by our group. Among the target genes of the inte
Publicado em: 2007
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4. The gamma 1(34.5) gene of herpes simplex virus 1 precludes neuroblastoma cells from triggering total shutoff of protein synthesis characteristic of programed cell death in neuronal cells.
The gamma 1(34.5) gene of herpes simplex virus 1 was previously shown to play a role in viral virulence since deletion of the gene reduced by a factor of approximately 100,000 the capacity of the virus to replicate in the central nervous system and cause mortality in the mouse. Here we show that in the human neuroblastoma cell line SK-N-SH of neuronal origin