Hypernatremia
Mostrando 13-19 de 19 artigos, teses e dissertações.
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13. Extracellular volume decreases while cell volume is maintained by ion uptake in rat brain during acute hypernatremia.
1. Regulation of brain extracellular and intracellular water content, regarded as volume, and electrolytes in response to 90 min of hypernatremia has been studied in the cerebral cortex of rats under urethane anaesthetic. 2. Total tissue electrolytes and water were partitioned between extracellular and intracellular compartments based on measurements made in
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14. Hypernatremia associated with cathartics in overdose management.
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15. Inhibition of Renal Tubular Sodium Reabsorption by Hypernatremia*
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16. Diffuse demyelinating lesions of the brain after the rapid development of hypernatremia.
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17. Sodium and brain injury: do we know what we are doing?
There is mounting evidence, including the recent report by Maggiore and colleagues, of an association between hypernatremia and mortality in patients with traumatic brain injury. This mandates a re-evaluation of routine administration of agents such as hypertonic saline for the management of intracranial hypertension in those with traumatic brain injury.
BioMed Central.
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18. Factors that limit brain volume changes in response to acute and sustained hyper- and hyponatremia
Rats were made acutely hyper- or hyponatremic by infusion of hypertonic saline or water, respectively. Other rats were maintained in these states from 1 to 7 days to observe the effects of time. Brain tissue water, Na, Cl, and K were compared with serum Na and Cl concentration (NaE and ClE). The following observations are noted: Brain Cl content varies direc
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19. “Essential” hypernatremia due to ineffective osmotic and intact volume regulation of vasopressin secretion
A physiological explanation for sustained hyperosmolality was sought in a patient with histiocytosis. During 23 days of observation with only sodium intake regulated at 100 mEq daily, elevation (mean 310 mOsm/kg of water) and fluctuation (range 298-323) of the fasting plasma osmolality were recorded. The presence of endogenous vasopressin was indicated by th