TOL plasmid can prevent induction of chemotactic responses to aromatic acids.
AUTOR(ES)
Harwood, C S
RESUMO
Growth conditions that elicited positive chemotaxis to benzoate and m-toluate in TOL- Pseudomonas putida cells failed to elicit taxis to these compounds in TOL+ cells. The inability of TOL+ cells to respond to these aromatic acids appears to be due to the preferential expression of TOL-encoded genes for aromatic degradation over chromosomally encoded genes. Expression of chromosomal genes for aromatic degradation is required for cells to form beta-ketoadipate, the inducer of benzoate and m-toluate taxis.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=214810Documentos Relacionados
- Chemotactic response of Escherichia coli to chemically synthesized amino acids.
- NAH plasmid-mediated catabolism of anthracene and phenanthrene to naphthoic acids.
- Electrophysiological responses of the chemoreceptors of the blowfly to sodium salts of fatty acids.
- Identification of cis-diols as intermediates in the oxidation of aromatic acids by a strain of Pseudomonas putida that contains a TOL plasmid.
- Macrophage response to bacteria: induction of marked secretory and cellular activities by lipoteichoic acids.
